CHARACTERIZATION OF COLE1-LIKE PLASMID PIP843 FROM KLEBSIELLA PNEUMONIAE ENCODING EXTENDED-SPECTRUM b-LACTAMASE CTX-M-17

 

CAO THI BAO VAN1,3 , THIERRY LAMBERT 1,2 , PATRICE COURVALIN 1

1 Unité des Agents Antibactériens, Institut Pasteur ,
2
Centre d’Etude Pharmaceutiques, Châtenay–Malabry, France, 3 Institut Pasteur d’Ho Chi Minh Ville, Viet Nam.

 

Resistance of Klebsiella pneumoniae BM4493 isolated in Ho Chi Minh City in Vietnam, to cefotaxime and aztreonam was due to production of the novel CTX-M-17 b-lactamase. The blaCTX-M-17 gene was borne by the 7,086-bp pIP843 plasmid which was entirely sequenced and belonged to the ColE1-family. The 876-bp blaCTX-M-17 differed from blaCTX-M-14 by two nucleotides that led to the single amino acid substitution Glu289®Lys. The blaCTX-M-17 gene was flanked upstream by a ISEcp1-like element and downstream by an IS903-variant designated IS903-C. The transcriptional start site of blaCTX-M-17 was located 109 nucleotides upstream from the initiation codon in the ISEcp1-like element, which also provided the promotor sequences. Plasmid pIP843 contained 5 ORFs transcribed in the same orientation. Regions homologous to sequences coding for putative RNAII, RNAI transcripts and a to a rom gene, involved in regulation of transcription and of stability of ColE1-like plasmids were identified. Consensus sequences for putative replication origin (oriV) and transfer (oriT) origins were present. Results of a transposition assay and of primer extension experiments indicate that ISEcp1 can contribute to dissemination of blaCTX-M-17 and also provides the promoter for expression of this gene.

The CTX-M enzymes represent a new family of class A extended-spectrum ß-lactamases (ESBL). The first member, MEN-1 (CTX-M-1), was reported at the beginning of the 1990s (3, 4). In contrast to TEM and SHV type cefotaxime-hydrolyzing ESBLs, CTX-M ß-lactamases are much more active against cefotaxime as a substrate than against ceftazidime. This probably results from changes in amino acids critical for extended-spectrum activity (3, 15, 18, 22).

To date, the fast growing CTX-M family comprises 14 members: CTX-M-1 (MEN-1) (3, 4), CTX-M-2 (5), CTX-M-3 (16), CTX-M-4 (13), CTX-M-5 (7), CTX-M-7 (previously designated CTX-M-5) (14), CTX-M-8 (6), CTX-M-9 (31), CTX-10 (27), CTX-M-13, 14, 15 (accession no. AF252621, AF252622, AF252623, unpublished data), Toho-1 (19), and Toho-2 (22). These enzymes have been mainly found in Escherichia coli and Salmonella isolated from South America (5), Eastern Europe (7, 16), and Japan (19, 22). Little is known about the environment of the structural genes for these enzymes and their promoters have not been identified experimentally.

Numerous bla genes have been found located on plasmids and some of them are part of transposons or constitute cassettes in integrons (23, 29). The aim of this work was to characterize the CTX-M-type ß-lactamase from Klebsiella pneumoniae BM4493 isolated in Viet Nam, which was resistant to cefotaxime and apparently susceptible to ceftazidime, and to identify its genetic basis.